Activity and function of the endothelial sodium channel is regulated by the effector domain of MARCKS-like protein 1 in mouse aortic endothelial cells
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In this study, we investigated the functional role of myristoylated alanine-rich C-kinase substrate-like protein 1 (MLP1) phosphorylation in regulating endothelial sodium channel (EnNaC) activity using mouse aortic endothelial cells for the first time. The results from this study will help elucidate the molecular mechanism by which aortic stiffness is regulated by EnNaC.
